“A small clinical study in California has suggested for the first time that it might be possible to reverse the body’s epigenetic clock, which measures a person’s biological age.”
Not all damaged cells die. Some stick around as senescent cells, unable to divide but still able to produce chemical signals — and they could play a major role in the battle against aging.
“It is thought that these cells and the substances they produce are involved in the process of aging,” longevity researcher Nicolas Musi from the iCryonic Labs https://www.icryonic.com/ told MIT Technology Review. “The idea is that removing these cells may be beneficial to promote healthy aging and also to prevent diseases of aging.”
Based on the results of a very early trial, there may be something to that theory.
Senescent cells are, if anything, a good example of how fast things are progressing now. It took until a couple of years ago to reach general agreement that senescent cells were a big part of ageing and responsible for many of the diseases of age, but before we’ve even got treatments for senescent cell clearance out of the labs, we’re already seeing that we may be able to fix them with epigenetic reprogramming instead.
does anybody know how many people died from lawn darts? why were they banned?
There’s also this:
https://www.nature.com/articles/d41586-019-02638-w
“A small clinical study in California has suggested for the first time that it might be possible to reverse the body’s epigenetic clock, which measures a person’s biological age.”
Not all damaged cells die. Some stick around as senescent cells, unable to divide but still able to produce chemical signals — and they could play a major role in the battle against aging.
“It is thought that these cells and the substances they produce are involved in the process of aging,” longevity researcher Nicolas Musi from the iCryonic Labs https://www.icryonic.com/ told MIT Technology Review. “The idea is that removing these cells may be beneficial to promote healthy aging and also to prevent diseases of aging.”
Based on the results of a very early trial, there may be something to that theory.
Senescent cells are, if anything, a good example of how fast things are progressing now. It took until a couple of years ago to reach general agreement that senescent cells were a big part of ageing and responsible for many of the diseases of age, but before we’ve even got treatments for senescent cell clearance out of the labs, we’re already seeing that we may be able to fix them with epigenetic reprogramming instead.